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Preeclampsia is a pregnancy complication characterized by high blood pressure and signs of damage to kidneys and liver. Preeclampsia usually starts after 20 weeks of pregnancy in women whose blood pressure had been normal. Slight rise in blood pressure may be a sign of preeclampsia.
Pregnant women risk of preeclampsia can be linked in some cases to genes from fetus. Preeclampsia usually starts with a problem in the placenta, which is mainly composed of fetal cells.
Researchers analysed a large number of women, and discovered several genetic variables that identified the same gene associated with the risk.
The genetic variants identified by the researchers are associated with the FLT-1 gene, which encodes a protein that, in soluble form, is known as a biomarker for preeclampsia.
The protein contributes to damaging the mother’s vascular system causing high blood pressure and an increased amount of protein in the urine. This happens as a result of problems with the placenta, essentially because fetal cells in the placenta produce the protein.
The risk of the disease may be because changes in the gene affect how the protein works and how it affects the mother. This is a fetal gene that gives the mother an increased risk of developing preeclampsia.
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