Posted by 
Inflammatory bowel disease IBD is a painful conditions that can cause severe diarrhea and fatigue. Taking a cup of strawberries daily could prevents inflammatory bowel disease. Sedentary lifestyle and diet high in sugar, animal-fat and in low-fiber may promote colonic inflammation and increase the risk of IBD.
IBD includes Crohn’s disease which can infect any part of the gastrointestinal tract, and ulcerative colitis, which is characterized by inflammation of the colon and rectum. Daily intake of fruits and vegetables has been associated with a lowered risk of IBD.
Researchers at the University of Massachusetts Amherst focused on strawberries due to their wide consumption. A group of healthy mice consuming a regular diet, and three groups of mice with IBD consuming a regular diet, a diet with 2.5 percent whole strawberry powder or a diet with 5 percent whole strawberry powder.
The abundance of pro-inflammatory immune cells was reduced by dietary WS in the colonic mucosa, which was accompanied by the suppression of abnormal overproduction of pro-inflammatory cytokines such as TNF-α, IL-1β, and IFN-γ in the colon of the colonic mice.
The researchers found that dietary consumption of whole strawberries at a dose equivalent to as low as three-quarters of a cup of strawberries per day in humans significantly suppressed symptoms like body weight loss and bloody diarrhea in mice with IBD.
Strawberry is an edible berry with various potential health benefits. Strawberry treatments also diminished inflammatory responses in the mice’s colonic tissue.
Colonic inflammation adversely impacts the composition of microbiota in the gut. With IBD, the abundance of harmful bacteria increases, while levels of beneficial bacteria decrease in the colon. Following the dietary treatments of whole strawberries, the researchers observed a reversal of that unhealthy microbiota composition in the IBD mice.
Dietary intake of the whole strawberry inhibited colonic inflammation, restored immune homeostasis and alleviated gut microbiota dysbiosis in dextran sulfate sodium-treated mice.